❓What do you think about this rhythm?
Anonymous Poll
8%
SVT with aberrancy
6%
Monomorphic VT
75%
Polymorphic VT
11%
Artifact / technical issue
❤8
ECG.CASES
❓What do you think about this rhythm?
This is a real arrhythmia, not an artifact. Careful review shows an irregular, continuously changing QRS morphology consistent with polymorphic VT.
The arrhythmia begins with a PVC falling on the preceding T wave (R on T phenomenon), even though the QT interval is normal.
Recognizing this pattern is crucial, as management differs entirely from a benign tracing or technical artifact.
The arrhythmia begins with a PVC falling on the preceding T wave (R on T phenomenon), even though the QT interval is normal.
Recognizing this pattern is crucial, as management differs entirely from a benign tracing or technical artifact.
❤11
What is the rhythm?
Anonymous Poll
9%
Atrial Fibrillation
14%
Atrial flutter with 2:1 conduction
15%
Atrial tachycardia
62%
AVNRT
❤17
What explains this finding?
Anonymous Poll
26%
Sinus pause
28%
Second-degree AV block
29%
Non-conducted PAC
18%
Sinus exit block
❤9👍7
What is the most likely diagnosis?
Anonymous Poll
17%
Hyperkalemia
34%
Sodium channel blocker toxicity
25%
Acute MI complicated by cardiogenic shock
24%
Ventricular tachycardia
👍7❤2
The ECG demonstrates sinus tachycardia, with subtle P waves merging into the preceding T waves (as shown in the image).
There is right axis deviation, QRS prolongation (>100 ms), and a distinct terminal R wave in aVR (also highlighted in the image).
The QT interval is prolonged as well.
Although ST elevation in aVR and V1 may raise concern for acute MI, and a wide QRS can occur in hyperkalemia, the overall ECG pattern, combined with the patient’s symptoms (decreased level of consciousness and severely low blood pressure) strongly suggests sodium channel blocker toxicity, most classically TCA overdose given the patient’s history of depression , or toxicity from certain class I antiarrhythmic agents.
There is right axis deviation, QRS prolongation (>100 ms), and a distinct terminal R wave in aVR (also highlighted in the image).
The QT interval is prolonged as well.
Although ST elevation in aVR and V1 may raise concern for acute MI, and a wide QRS can occur in hyperkalemia, the overall ECG pattern, combined with the patient’s symptoms (decreased level of consciousness and severely low blood pressure) strongly suggests sodium channel blocker toxicity, most classically TCA overdose given the patient’s history of depression , or toxicity from certain class I antiarrhythmic agents.
❤23